Publication for INHBC and INHBE
| Species | Symbol | Function* | Entrez Gene ID* | Other ID | Gene coexpression |
CoexViewer |
|---|---|---|---|---|---|---|
| hsa | INHBC | inhibin subunit beta C | 3626 | [link] | ||
| hsa | INHBE | inhibin subunit beta E | 83729 |
| Pubmed ID | Priority | Text |
|---|---|---|
| 21407220 | 0.98 | INHBC and INHBE mRNA during liver regeneration after partial hepatectomy, or chemical injury in rodents produced conflicting data on possible biological functions as reviewed recently. |
| 0.98 | INHBC and INHBE are predominantly, but not specifically expressed in the liver as assumed previously. | |
| 0.97 | INHBC and INHBE). | |
| 0.97 | INHBE, whereas INHBC expression remained unaltered. | |
| 0.97 | INHBC and INHBE expression were at least 100-fold higher in liver than in any other organ investigated, but nevertheless detected in 11 out of 13 and 9 out of 13 tissues, respectively. | |
| 0.96 | INHBC and INHBE expression levels are highest in the liver, much lower in testis, adrenal and pituitary gland and almost undetectable in other organs of rodents. | |
| 0.96 | INHBC and INHBE was increased in some and decreased in other HCC, overall no significant changes were noted. | |
| 0.95 | INHBC and INHBE were highly expressed in all three species, with INHBC levels being about 10-fold higher in rodent than in human liver. | |
| 0.91 | INHBC from 26.0 to 38.5 and for INHBE from 24.5 to 36.5. | |
| 20209104 | 0.97 | INHBC and INHBE genes, encoding betaC and betaE) (Fig. 1B). |
| 21037107 | 0.97 | INHBE, INHBC), both of which were up-regulated with HRT. |
| 26317806 | 0.96 | INHBC), ras homolog family member A(RHOA), latent transforming growth factor beta binding protein 1 (LTBP1), SMAD family member 5 (SMAD5), SMAD family member 9 (SMAD9), retinoblastoma-like 1 (p107) (RBL1), transcription factor Dp-1(TFDP1), THBS1, growth differentiation factor 5 (GDF5), inhibin, beta E(INHBE),chordin(CHRD) andring-box 1, E3 ubiquitin protein ligase(RBX1). |
| 18053128 | 0.95 | INHBC and INHBE originated by a duplication event in tetrapod lineage after its divergence from bony fishes. |
| 0.94 | INHBE, INHBC and SLC4A8, SLC4A10 genes arose after the fish-tetrapods split. | |
| 23200667 | 0.95 | INHBC and INHBE for stem cell signaling regulation. |
| 20876727 | 0.92 | INHBC, and INHBE gene products, respectively. |
| 31024340 | 0.88 | INHBE, INHBC), proteoglycan 4 (PRG4), serum amyloid A-4 protein precursor (SAA4), and vitronectin (VTN). |
| 23263486 | 0.87 | INHBC-INHBE and ACVR2A) were connected with three genes implicated by the functional association network analysis (ACVR1B-ACVRL1, ACVR1C and BMPR2), one of which replicated (Supplementary Fig. 8). |
| 25344915 | 0.82 | INHBC, INHBE, MSTN, GDF15 and TGFB3), that plays a role in cell migration, invasion and epithelial-mesenchymal transition (EMT). |
| 30268436 | 0.80 | INHBC and INHBE were highly expressed in LIHC, whereas BMP3 and BMP5 were highly expressed in LUAD (Figure 5B). |
| 0.66 | INHBC, INHBE, INHBA, and TGFB3. |
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