Publication for IFIT3 and STAT1
| Species | Symbol | Function* | Entrez Gene ID* | Other ID | Gene coexpression |
CoexViewer |
|---|---|---|---|---|---|---|
| hsa | IFIT3 | interferon induced protein with tetratricopeptide repeats 3 | 3437 | [link] | ||
| hsa | STAT1 | signal transducer and activator of transcription 1 | 6772 |
| Pubmed ID | Priority | Text |
|---|---|---|
| 25905045 | 0.96 | IFIT3, Mx1, and STAT1) and either anti-viral responses or virus-host interactions (AP2A1, CH60, DDX58, ELOA1, IFIT1, IFIT3, ISG15, Mx1, RBP2, and STAT1). |
| 25650658 | 0.95 | IFIT3 mRNA depending on stimulation with IFN and blockade of NFkappaB and STAT1 signaling |
| 0.75 | IFIT3 showed binding to JNK and STAT1, the latter being an important inducer of IFIT3 expression. | |
| 0.63 | Signal Transducers and Activators of Transcription 1 (STAT1) and IFN-induced protein with tetratricopeptide repeats 3 (IFIT3) were significantly increased in L3.6pl. | |
| 23623029 | 0.94 | IFIT3, MX1, STAT1, IFI44L, USP18, TRIM22 and BST2 among others (Table II). |
| 0.89 | IFIT3, MX1, STAT1, IFI44L, USP18, TRIM22 and BST2, suggesting epigenetic transcriptional accessibility in these genetic loci. | |
| 30158934 | 0.94 | STAT1 and then subjected to Q-PCR using primers to ISRE of the promoter of Ifit1 (C) or Ifit3 (D). |
| 31921100 | 0.94 | IFIT3 were as highly elevated in the absence of STAT1-mediated IFNAR signaling as in the parental HFs underlining the capacity of such IFN-independent mechanisms to profoundly regulate ISG expression. |
| 30177931 | 0.92 | Ifit3, Isg20, Atg7, Adrm1, and Stat1 (Figure 7). |
| 0.86 | Ifit3, Isg20, Atg7, Adrm1 and Stat1. | |
| 21886832 | 0.90 | STAT1 (4.27 fold in the absence of HCV to 2.29 fold in the presence of HCV, P = 4.04x10-4), as well as 5 of 7 genes downstream of STAT1 (IFIT1, IFIT3, IRF1, MX1, and TAP1). |
| 24065129 | 0.89 | ISGF3 and its target genes, a subset of ISGs (IFI27, OAS2, MX1, BST2, IFIT1, and IFIT3), which were previously found to be induced by U-STAT1. |
| 0.83 | -STAT1 in BJ cells upregulated the expression of the IKKe-independent genes IFIT1, IFIT3, and G1P2 but did not change expression of the IKKe-dependent genes ADAR and IFIT2. | |
| 0.78 | -STAT1, U-STAT2, and IRF9 together in hTERT-HME1 cells significantly increased the expression of the target genes, IFI27, OAS1, OAS2, MX1, IFIT1, and IFIT3 (Figure 2C), indicating that STAT1 tyrosine phosphorylation was not involved. | |
| 0.62 | ISGF3-induced genes (IFI27, BST2, OAS2, MX1, IFIT1, and IFIT3) and other ISGs (MYD88, IFI16, ADAR, and IRF1) was examined by real-time PCR. | |
| 30283459 | 0.81 | STAT1 targets; IFIT1, IFIT2, IFIT3, OAS1, OAS2, MX1, MX2, ISG15 as STAT1-STAT2 targets; SOCS3, CCND1, MMP3, FAS PIM1, VEGF, S1PR1 as STAT3 targets). |
| 0.76 | STAT1 targets; IFIT1, IFIT2, IFIT3, OAS1, OAS2, MX1, MX2, ISG15 as STAT1-STAT2 targets; SOCS3, CCND1, MMP3, FAS PIM1, VEGF, S1PR1 as STAT3 targets) could be recognized. | |
| 28152048 | 0.77 | STAT1 and STAT2), and multiple proteins directly involved in restriction of viral replication (IFIT1, IFIT3, and viperin) (Fig 6C). |
| 0.59 | STAT1, IFIT1, IFIT3, viperin) following infection with PR-2015 at MOIs of 1 and 10. | |
| 28471454 | 0.71 | STAT1 and its direct target genes IFIT3 and CXCL10 were markedly upregulated and prolonged by Dip G. It has been reported that STAT1 induction and activation have an important role in myeloid differentiation mainly through the regulation of a subset of IFN-gamma-inducible genes, such as the upregulation of CXCL10 and the downregulation of c-Myc. |
| 0.59 | STAT1, IFIT3 and C-X-C motif chemokine ligand 10 (CXCL10) was detected using quantitative real-time RT-PCR analysis. | |
| 30186768 | 0.70 | STAT1, IFI44, IFIT3, OAS1, IFIT1, ISG15, MX1, and USP18, was also shown to predict poor outcomes in glioblastomas post RT. |
| 24223959 | 0.69 | STAT1 or STAT3 reduced DV-induced IFIT3 expression. |
| 0.68 | IFIT3 in DV-infected A549 cells with knockdown of either STAT2 (B), STAT1 (C) or STAT3 (D) was determined by western blotting (B, C, and D) or quantitative RT/PCR (B). | |
| 0.58 | STAT1 or STAT3, regulated DV-induced IFIT3 production. | |
| 0.56 | STAT1 or STAT3 did not affect DV-induced IFIT3 expression (Figures 2C and 2D). | |
| 19752753 | 0.68 | IFIT3 in response to IFN-alpha2c, but Stat1-RNAi did not. |
| 26479788 | 0.68 | IFIT3, and OASL) mRNA in indicated U3A-STAT1 cell lines with or without IFN-beta (10 U/ml) for 12 hours. |
| 23825585 | 0.67 | STAT1 on gene transcription regulation, the expressions of two direct STAT1 target genes (RIG-G and CXCL-10) were tested by real time PCR. |
| 27780205 | 0.65 | IFIT3, MXA, and OAS1 was actually induced by semiphosphorylated STAT1:STAT2 heterodimers, albeit in association with IRF9. |
| 32048448 | 0.51 | STAT1, MX1, OAS1, SOCS1, STAT2, TAP1, IFI6, IFI35, IFIT1, IFIT3, IFITM1, IFITM3, ISG15 and JAK2. |
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