Publication for HNRNPD and NCL

Species	Symbol	Function*	Entrez Gene ID*	Other ID	Gene coexpression	CoexViewer
hsa	HNRNPD	heterogeneous nuclear ribonucleoprotein D	3184			[link]
hsa	NCL	nucleolin	4691

Pubmed ID	Priority	Text
27575817	0.98	AUF1 (p<0.001), HuR (p<0.001), TIA (p<0.001), TIAR (p<0.05) and NCL (p<0.001) and RECK (p<0.001) in BA cells from the patients with pulmonary sarcoidosis compared to those in healthy controls (Fig 1 and S3 Table).
	0.97	AUF1, TIA and NCL) in bronchoalveolar (BA) cells from our patients with pulmonary sarcoidosis compared to those from our healthy controls.
	0.97	AUF1, NCL and mainly TIA are important regulation factors namely in pulmonary interstitial lung diseases.
	0.95	AUF1, NCL and TIA) may not be associated with sarcoidosis but rather be an effect of the elevated sub-population of BA lymphocytes being a hallmark of pulmonary sarcoidosis.
	0.94	AUF1 (p<0.001), TIA (p<0.001), NCL (p<0.01) and RECK (p<0.05) was decreased in sarcoidosis compared to healthy controls.
	0.94	AUF1, TIA and NCL in our patients with sarcoidosis is independent of smoking status.
	0.89	AUF1 (p = 0.002), NCL (p = 0.02), HuR (p = 0.002), TIA (p<0.001) and TIAR (p = 0.007).
	0.85	AUF1, TIA and NCL) in unseparated bronchoalveolar cells and the down-regulated protein expressions of two RBPs (AUF1 and HuR) in peripheral blood T lymphocytes were observed in our patients with pulmonary sarcoidosis.
	0.84	AUF1, TIA, NCL and addition TIAR and PCBP2) to be decreased in our patients with IIPs, but not in our patients with airway obstructive pathologies (COPD and asthma) when comparing to healthy controls.
	0.84	AUF1, HuR, NCL, TIA and PCBP2).
	0.77	AUF1, HuR, NCL, TIA, TIAR, PCBP2) and two inhibitors of proteolytic enzymes (RECK, PTEN) in pulmonary sarcoidosis and compared it to the expression in four control groups of healthy individuals and patients with other respiratory diseases: chronic obstructive pulmonary disease (COPD), asthma and idiopathic interstitial pneumonias (IIPs).
28624205	0.98	NCL, AUF1, and HuR protein expression.
	0.75	NCL has four RNA-binding domains and can stabilize its targeted mRNA, whereas AUF1 is able to bind to AU-rich element (ARE)-mRNAs and promote its targeted mRNA degradation.
26918608	0.97	nucleolin (NCL), HUR, and AUF1, have been reported to bind their target mRNA and modulate mRNA stability.
	0.97	NCL and AUF1 protein expression were observed in p85alpha-/- cells as compared with those in p85alpha+/+ cells.
	0.53	NCL, and AUF1 were also reduced in p85alpha-/- cells (Figure 3B).
31016112	0.97	HNRNPD, ELAVL1, and nucleolin (NCL) (which are able to bind to many ARE-mRNAs and regulate mRNA stability) were tested and compared.
	0.95	HNRNPD in vitro cultured UMUC3 cells, HNRNPD protein expression was markedly increased in xenograft tumor tissues attained from nude mice injected with UMUC3(shATG7) cells (Figure 6E,F), and the similar change of NCL protein expression (Figure S3D,E, Supporting Information).
	0.83	NCL, ELAVL1, and HNRNPD protein expression.
25014217	0.97	AUF1, nucleolin, HUR, ZFP36L1) that are now known to target AREs in the BCL2 3' UTR are themselves capable of physical association with each other via protein-protein interaction (see ref).
	0.95	AUF1 functions to destabilise BCL2 mRNA, nucleolin (acting antagonistically with AUF1) stabilises the BCL2 mRNA while HUR acts to both stabilise and enhance translational efficiency of BCL2 mRNA (reviewed in).
31071529	0.97	NCL, HuR, and AUF1 protein expression levels.
	0.59	NCL proteins were comparable between T24T (miR-146b inhibitor) and T24T (nonsense) cells, AUF1 was elevated in miR-146b inhibition cells.
21622178	0.97	nucleolin can also block AUF1-dependent suppression of bcl-2 expression, but does so by competing for the AUF1 binding site in the bcl-2 mRNA 3'UTR.
23117626	0.95	AUF1, and nucleolin in senescent cells was previously reported.
	0.94	AUF1, nucleolin or TTP (not shown); instead, we focused on NF90.
	0.62	AUF1, NCL and NF90 in whole-cell lysates prepared from human diploid WI-38 fibroblasts ['young', proliferating (P, pdl 20) or senescent (S, pdl 54)] and from human IDH4 fibroblasts [P (+Dex) or S (-Dex for 9 days)]; the levels of GAPDH were assessed as a loading control.
23131833	0.95	AUF1 and nucleolin are binding partners in uninfected cells, the interaction of nucleolin with the human rhinovirus 5' NCR may be the result of an indirect binding event mediated by AUF1.
	0.94	AUF1 isoforms have been reported to interact with PCBP1 and PCBP2, nucleolin, and PABP, which have been demonstrated to interact with the 5' NCR, the 3' NCR, and the 3' poly(A) tract of poliovirus RNA, respectively.
30181254	0.95	AUF1 (p37, p40, p42, and p45) are labeled, and nucleolin was used as a loading control.
31417248	0.94	nucleolin, and AUF-1 could possibly influence LIF and require investigation in COPD models and tissue samples.
31877708	0.93	HNRNPD, HNRNPA1, SSB, SYNCRIP, NCL, and PCBP2; cluster III with XRCC6, PCNA, UBE2V2, UBE2K, and UBE2D3; cluster IV with EIF3G, TUFM, RPLP0, RPLP2, and ABCE1; cluster V with ATP1B1, ATP1A3, ATP1B3, and ATP1B3; cluster VI with ECHS1, HSD17B10, HADH, and ALDH2; cluster VII with CYCS, LCP1, ACTC1, ACTA2, ACTG2, CTTN, and GSN; and cluster VIII with RAB25, RAB2B, RAB11A, SEC22B, COPG1, COPG2, CD59, DYNC1I2, DYNLL1, RAB1A, and RAB6A.
30118841	0.88	NCL, HNRNPD and ELAVL1 were evaluated.
	0.62	NCL, HNRNPD and ELAVL1, have been reported to bind to their targeted mRNA and regulate their mRNA stability.
	0.51	NCL or HNRNPD.
15743974	0.87	nucleolin are implicated in RNA/DNA helicase activity and RGG-boxes in hnRNPK and hnRNPD and nucleolin are required for transcriptional activation.
21737422	0.87	nucleolin, but neither hnRNP D (also known as AUF1) nor hnRNP C1/C2 were detected in the nucleolin IP samples.
26584240	0.53	HNRNPD, NCL, and STAU1.
25402510	0.51	AUF1, HuR and nucleolin were tested in T24 and T24T cells.