Publication for FOXL1 and FOXC2

Species	Symbol	Function*	Entrez Gene ID*	Other ID	Gene coexpression	CoexViewer
hsa	FOXL1	forkhead box L1	2300			[link]
hsa	FOXC2	forkhead box C2	2303

Pubmed ID	Priority	Text
22567372	0.98	FOXC2, and FOXL1.
	0.95	FOXC2 and FOXL1 (Figure 1(d)).
	0.93	FOXC2 and FOXL1 genes, as a patient with a downstream deletion encompassing just FOXC2 and FOXL1 exhibited cardiac defects without any respiratory complications.
31910858	0.98	FOXC2 has been implicated in triple-negative breast cancer progression and therapy resistance, while FOXL1 is reported to inhibit breast cancer cell proliferation, invasion, and migration.
	0.96	FOXC2, FOXL1, and FOXC2-AS1)
	0.94	FOXC2 and FOXL1 are members of the Forkhead family of transcription factors with important functions in biological processes such as cell cycle control, proliferation, and development.
19500772	0.97	FOXC2, and FOXL1:as well as MTHFSD (genomic deletions).
	0.97	FOXC2, and FOXL1, showing the extent and gene content of the regions deleted in seven patients analyzed by array CGH (D1-D7).
	0.97	FOXC2 and FOXL1 and an intact FOXF1, is also shown.
	0.96	FOXC2 and FOXL1 did not.
	0.95	FOXC2 or FOXL1 may also contribute to this phenotype in humans, despite the fact that, in mice, haploinsufficiency for Foxf1 alone is associated with esophageal atresia.
	0.82	FOXC2 (MIM 602402) and FOXL1 (MIM 603252) genes at 16q24.1 have not only ACD/MPV, as expected, but also distinct malformations comprising congenital heart defect, in particular hypoplastic left heart syndrome, and gastrointestinal atresias, including esophageal atresia, as well as urinary tract malformations and other malformations.
	0.81	FOXC2 and FOXL1 but not FOXF1, patient D7, did not.
22407726	0.97	FOXC2/FOXL1 gene cluster and C16ORF95.
	0.97	FOXC2/FOXL1 loci.
	0.97	FOXC2, and FOXL1.
	0.84	FOXC2 and FOXL1.
25472632	0.97	FOXC2, and FOXL1.
19801982	0.96	FOXC2; FOXL1]
	0.90	FOXC2[602402],FOXL1[603252]; CRHR1[122561]; SPTBN1[182790]; MEPE[605912],IBSP[147563],SPP1[166490]; ARHGAP1[602732],LRP4[604270],F2[176930]; HDAC5[605315],C17orf53[None]; ZBTB40[612106]; ESR1[133430], C6orf97[None]; TNFRSF11B[602343]; LRP5[603506]; SP7[606633]; TNFSF11[602642],AKAP11[604696]; TNFRSF11A[603499].
	0.74	FOXL1) or are involved in adipocyte metabolism and early stage chondrogenic differentiation (FOXC2).
17062144	0.96	foxc2-foxl1 cluster appears broken, and foxc2 appears to have been lost.
	0.82	FoxC2-FoxL1 genes.
21315191	0.96	FOXC2, FOXL1 and MTHFSD, have more typical features of VACTERL association (including vertebral anomalies, gastrointestinal atresias, urinary tract malformations, and cardiac anomalies).
31367258	0.95	FOXC2, FOXF1 and FOXL-1.
	0.95	FOXC2, FOXF1 and FOXL-1were detected in CF129-knockdown PANC-1 and BxPC-3 cells by qRT-PCR.
	0.92	FOXC2, FOXF1 and FOXL-1 in CF129-overexpressing PANC-1/BxPC-3 cells.
28283553	0.95	FOXC2 (forkhead box protein C2) and FOXL1 (forkhead box protein L1) (Supplementary Figure 1).
	0.94	FOXC2 and FOXL1.
23565379	0.94	FOXC2, HOXC6, LRP4, MEF2C, PTHLH, RSPO3, SFRP4, TGFBR3, WLS, WNT3, WNT4, WNT5B, WNT16), bone development and ossification (CLCN7, CSF1, MEF2C, MEPE, PKDCC, PTHLH, RUNX2, SOX6, SOX9, SPP1, SP7), mesenchymal-stem-cell differentiation (FAM3C, MEF2C, RUNX2, SOX4, SOX9, SP7), osteoclast differentiation (JAG1, RUNX2), and TGF-signaling (FOXL1, SPTBN1, TGFBR3) (reviewed in.
20425471	0.92	FOXC2, and FOXL1.
27997041	0.92	FOXC2, FOXL1, and FOXF1.
31888141	0.91	FOXC2, and FOXL1 are associated with CHD, particularly HLHS.
28608148	0.88	FOXC2, FOXL1, GJA1, HAND1, HEY1, HEY2, HUWE1, IRX4, JAG1, MED20, MLL2, MYH6, NKX2-5, NOTCH1, NOTCH2, PTCH1, TBX2, TBX5 and USP44, and found 41 potential single nucleotide variants of which 8 were nonsynonymous changes (Figure 3A; Table 2).
	0.51	FOXC2, FOXL1, GJA1, HAND1, IRX4, MYH6, NKX2.5, NOTCH1 and TBX5.
31281520	0.84	FOXC2, FOXG1, FOXI1, FOXJ1, FOXJ2, FOXL1, FOXL2, FOXN1, FOXN4, and FOXO3 showed no obvious changes in CRC tissues compared with those in normal tissues (Figure S1-S2).
28355270	0.82	FOXC2, FOXL1, FOXB1, DDIT3 and others, can bind to these sites in different combinations (Fig 8E).