Publication for Pax2 and Tfap2b

Species	Symbol	Function*	Entrez Gene ID*	Other ID	Gene coexpression	CoexViewer
mmu	Pax2	paired box 2	18504			[link]
mmu	Tfap2b	transcription factor AP-2 beta	21419

Pubmed ID	Priority	Text
28912684	0.98	Tfap2B, has the ability to direct specification of Pax2+ GABAergic interneurons.
	0.98	Tfap2B in the VZ of the developing cerebellum at E12.5 results in a decrease of Pax2 expression, indicating that Tfap2B is necessary for the specification of cerebellar GABAergic interneurons.
	0.98	Tfap2B results in a decrease in Pax2 expression (Aiii).
	0.98	Tfap2B downstream of Ptf1a play distinct roles to ensure generation of Pax2+ interneurons.
	0.97	Tfap2B are now also expressing Pax2, and that, perhaps, Tfap2A+ and Tfap2B+ cells become GABAergic interneurons expressing Pax2 as they mature in the WM.
	0.97	Tfap2B, but not Tfap2A, results in a reduction in Pax2 expression.
	0.96	Tfap2B expression precedes Pax2 in the developing cerebellum.
	0.96	Tfap2B in directing the specification of GABAergic neuronal subtypes, we analyzed consequences of misexpressing each transcription factor on the expression of neuronal markers, Pax2, Pax6 and CBP which correspond to GABAergic interneurons, glutamatergic neurons and Purkinje cells, respectively.
	0.95	Tfap2B precedes the expression of Pax2 and raise the possibility that Tfap2A and Tfap2B regulate the expression of Pax2.
	0.95	Tfap2B to change the number of Pax2+ interneurons, we found a two-fold increase in the number of Pax2+ interneurons in Tfap2A-misexpressed cells relative to control in the WM (Figures 6A,B,D).
	0.93	Tfap2B are expressed by mitotic or postmitotic GABAergic neurons, we assessed whether these cells express transcription factors Lhx1/5 (postmitotic), Olig2 and Pax2 (both mitotic and postmitotic; Maricich and Herrup,; Chizhikov et al.,; Morales and Hatten,; Zhao et al.,).
	0.91	Tfap2B is sufficient to regulate Pax2 expression.
	0.91	Pax2+ interneurons in Tfap2B-knockdown cells in the WM (Figures 7A,C,D).
	0.90	Pax2+ neuronal population indicates that most Pax2+ interneurons express Tfap2A (E15.5: 97 +- 1%, E18.5: 89 +- 4%) and Tfap2B (E15.5: 88 +- 4%, E18.5: 90 +- 2%) at both E15.5 and E18.5.
	0.90	Tfap2B on the expression of Pax2, Pax6 and CBP at E15.5.
	0.78	Pax2 expression, while Tfap2B misexpression results in a decrease in the expression of Pax6.
	0.70	Tfap2B+ cells also overlap with Pax2 indicating that these cells identify for interneuron precursors (Supplementary Figure S3).
	0.63	Tfap2B subpopulations (Tfap2B+/Pax2+ and Tfap2B+-alone) made up of only interneurons, but not Purkinje cells, consistent with the lack of Tfap2B expression in Purkinje cells in the adult cerebellum (Figures 4D-F,J-L, see also Figures 2J-L).
24507192	0.98	Pax2+ / AP2Beta+), and perinatal olfactory bulb.
31566562	0.95	Pax2), and neural crest (Pax3, Hoxa2, Tfap2b and Neurog1) indicated mild neural and severe neural crest differentiation defects (Figure 6E and Figure 6:figure supplement 2).
23616538	0.92	Pax2 and Kcnip2, Tfap2b, Prkcq and Klhl14 in dorsal horn neurons.
	0.62	Pax2, but also several genes that had previously not been attributed to spinal inhibitory interneurons (e.g. Klhl14, Rorb, and Tfap2b).
	0.60	Tfap2b to display a high degree of overlap with Pax2 (Fig 5).