Publication for Hmgcr and Hsd17b7

Species	Symbol	Function*	Entrez Gene ID*	Other ID	Gene coexpression	CoexViewer
mmu	Hmgcr	3-hydroxy-3-methylglutaryl-Coenzyme A reductase	15357			[link]
mmu	Hsd17b7	hydroxysteroid (17-beta) dehydrogenase 7	15490

Pubmed ID	Priority	Text
26067871	0.98	Hmgcr, Hsd17b7, and Dhcr24) and transport (Abca1 and Abcg1) of cholesterol were up-regulated.
27878435	0.98	HMGCR, HSD3B7, HMGCS1, LSS, FDFT1, DHCR7, HSD17B7, NSDHL, DHCR24, FDPS, SIGMAR1, SQLE, MVK, that are expressed in the lens.
28150810	0.98	Hmgcr, Idi1, Sqle, Cyp51, Msmo1, Hsd17b7, and Dhcr24) were among the genes down-regulated in XX/Sry Sertoli cells, suggesting that the down-regulation of the cholesterogenic genes was primarily the result of the decreased expression of Srebf2.
28720595	0.97	Hmgcr, Sqle, Scd5, Hsd17b7 and Dhcr7, are upregulated.
30069000	0.97	Hmgcr, Hmgcs1, Hsd17b7, Idi1, Lss, Mvd, Mvk, Msmo1, Nsdhl, Pmvk, Sc5d, Sqle, and Tm7sf2 were significantly decreased in HFD livers.
31296899	0.97	Hmgcr, Mvd, Hsd17b7, and Dhcr7) and steroid hormone synthesis (StAR, StARD3, and Hsd17b7), but downregulation of the genes driving TG synthesis from the lists of GSEA (Fig. 3E, Supplementary Fig. 5).
26921468	0.83	HMG-CoA reductase, which would in turn cause SREBP2-mediated up-regulation of most enzymes involved in cholesterol biosynthesis, including residual activity of Hsd17b7; and 2) dietary supplementation with cholesterol (standard diet + 2% cholesterol by weight) to cause negative feedback on the pathway by providing its end product (Fig. 7A).