Publication for Gfi1b and Nfe2
| Species | Symbol | Function* | Entrez Gene ID* | Other ID | Gene coexpression |
CoexViewer |
|---|---|---|---|---|---|---|
| mmu | Gfi1b | growth factor independent 1B | 14582 | [link] | ||
| mmu | Nfe2 | nuclear factor, erythroid derived 2 | 18022 |
| Pubmed ID | Priority | Text |
|---|---|---|
| 29472540 | 0.98 | NFE2 (may promote CTCF binding) and GFI1b motifs (may reduce CTCF binding) relative to constitutive CTCF peaks (Fig. 5d). |
| 0.97 | NFE2, TAL1, KLF1, and GFI1b. | |
| 0.83 | GFI1b and NFE2 motifs are enriched in erythroid-specific CTCF peaks (foreground) relative to constitutive CTCF peaks (background) both when considering all CTCF peaks and the subset that contain a CTCF motif discSNP as in Fig. 5c. | |
| 29861167 | 0.98 | NFE2, and GFI1B genes and their regulons, was the most stable core of response across all hemogenic subsets. |
| 0.86 | NFE2, KLF1, and GFI1B regulons were not active, while, SOX17 and SOX18 regulons displayed slight increases in their regulon-level signal in non-HE. | |
| 23378057 | 0.97 | Gfi1b, Nfe2, Gata2, and Ldb1 that have specific regulatory regions occupied by Ldb1 in ChIP-seq analysis in HSPCs (Figure S1). |
| 23524953 | 0.97 | Nfe2, Eto2, Gfi1b, Gata1 and Ldb1) known to be important in the erythroid/megakaryocytic lineages, while there was a negative correlation between PU.1 and Gata1, which are thought to function as a switch controlling erythroid and myelomonocytic fates. |
| 28973433 | 0.97 | Gfi1b, and Nfe2, whereas CD41+, CD45+ cells exhibited higher levels of Spi1 and Cebpb (Supplementary Figure S1). |
| 26954547 | 0.96 | Gfi1b, Myb, Cebpalpha, Pu.1, Nfe2 and Ikzf1). |
| 25664528 | 0.71 | Nfe2, Gfi1b, Ikzf1 (Ikaros) and Myb), and low expression of endothelial genes (Erg, Sox7, Sox17, Hoxb4, Cdh5). |
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