Publication for G6pc and Aldob
| Species | Symbol | Function* | Entrez Gene ID* | Other ID | Gene coexpression |
CoexViewer |
|---|---|---|---|---|---|---|
| mmu | G6pc | glucose-6-phosphatase, catalytic | 14377 | [link] | ||
| mmu | Aldob | aldolase B, fructose-bisphosphate | 230163 |
| Pubmed ID | Priority | Text |
|---|---|---|
| 30158026 | 0.98 | AldoB, TrioK, Ldh), and gluconeogenic enzymes (G6pc and Fbp1) in the intestine (Fig. 3). |
| 0.98 | AldoB, Triok, and Ldh), and gluconeogenesis (G6pc and Fbp1) are significantly increased in the intestinal cells. | |
| 0.94 | Aldolase B, Triokinase), gluconeogenesis (G6pase, Fbp1), and hormone and hormone receptors (fibroblast growth factor 21, glucagon receptor, and adiponectin receptor 2). | |
| 29545180 | 0.98 | G6pc-/- mice, restoration of hepatic G6Pase-alpha expression in L-G6pc-/- mice normalized hepatic levels of c-Myc and c-Myc-regulated glycolytic enzymes, including GPI and aldolase B (Fig. 3A). |
| 0.98 | aldolase B and GPI (Fig. 4A) as wells as lactate (Fig. 4B), markers of glycolysis were increased in L-G6pc-/- mice at both pre-tumor and tumor-developing stages. | |
| 26558285 | 0.76 | Aldob (4.39), Aldoc (1.57), Bpgm (1.62), Eno1 (3.42), Eno2 (1.51), Eno3 (3.00), Galm (2.13), Gapdhs (3.73), Gck (1.50), Pgam2 (1.50), Pgk2 (1.58), Pgm1 (1.88), Pgm2 (1.98), Pgm3 (1.68), Pklr (1.61), and Tpi1 (1.86)], gluconeogenesis [Fbp1 (2.85), Fbp2 (3.16), G6pc (4.55), and Pck2 (2.03)], the TCA cycle [Acly (4.50), Aco1 (2.64), Aco2 (3.78), Cs (3.96), Dlat (3.21), Dld (2.75), Dlst (2.61), Idh1 (1.98), Idh2 (1.90), Idh3a (2.85), Pck2 (2.03), Pdha1 (2.25), and Pdhb (2.76)], pentose phosphate pathway (PPP) [G6pdx (3.70), Rbks (2.11), Rpe (2.04), and Tkt (2.01)], and glycogen metabolism [Gbe1 (2.57) and Ugp2 (2.39) for synthesis and Agl (3.24), Pgm1 (1.88), Pgm2 (1.98), Pgm3 (1.68), and Pygm (2.07) for degradation] was found to be significantly upregulated in Cmah-null mouse livers compared to controls (Figure 3(c)). |
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