Publication for COL1A2 and COL11A1

Species	Symbol	Function*	Entrez Gene ID*	Other ID	Gene coexpression	CoexViewer
hsa	COL1A2	collagen type I alpha 2 chain	1278			[link]
hsa	COL11A1	collagen type XI alpha 1 chain	1301

Pubmed ID	Priority	Text
30723390	0.97	COL1A2, COL11A1, MMP2, PXDN, BGN, COL5A1, COL8A1, and TGFB1I1 indicated poor prognosis of the patients(P < 0.05).
	0.97	COL1A2, COL11A1, MMP2, PXDN, BGN, and THY1(Fig. 5).
	0.97	COL1A2, COL11A1, MMP2, PXDN, and THY1 was related to the poor prognosis of the disease.
	0.97	COL1A2, NTM, COL11A1, THBS2, COL8A1, COL1A1, BGN, MMP2, PXDN, THY1, and TGFB1I1.
	0.97	COL1A2, COL11A1, MMP2, PXDN, BGN, COL5A1, COL8A1, and TGFB1I1 were associated with the patient's prognosis(P < 0.05).
	0.97	COL1A2, COL11A1, MMP2, PXDN, BGN, COL5A1, COL8A1 associated with the tumor stage as well as tumor patients' prognosis.
	0.96	COL1A2, COL11A1, MMP2, PXDN, BGN, COL5A1, COL8A1 associated with the tumor stage as well as tumor patients' prognosis.
	0.96	COL1A2, (f) COL11A1, (g) MMP2, (h) PXDN, and (i) THY1.
	0.92	COL1A2, COL1A1, COL11A1, THBS2, COL5A1), protein digestion and absorption(hsa04974)(COL3A1, COL6A3, COL1A2, COL1A1, COL11A1, COL5A1),focal adhesion(hsa04510)(COL3A1, COL6A3, COL1A2, COL1A1, COL11A1, THBS2, COL5A1),and PI3K-Akt signaling pathway(hsa04151)(COL3A1, COL6A3, COL1A2, COL1A1, COL11A1, THBS2, COL5A1) in bladder cancer.
	0.78	COL1A2, NTM, COL11A1, THBS2, COL8A1, COL1A1, BGN, MMP2, PXDN, THY1, and TGFB1I1.
26870242	0.97	COL1A2, COL6A3, THBS2, COL11A1, PDLIM7 and ADARB1 were involved in the progression of GC.
	0.97	COL1A2, COL1A1, THBS2 and COL11A1were identified to be involved in the ECM-receptor interaction pathway.
	0.91	COL1A2, COL1A1, COL5A2, THBS2, COL11A1 and COL5A1) were involved in the ECM-receptor interaction pathway.
	0.63	COL1A2, COL1A1, COL5A2, thrombospondin 2 (THBS2), COL11A1 and COL5A1] were predicted to participate in the pathway.
32058995	0.97	COL1A2, COL1A1, COL11A1, COL4A6, and THBS2) are involved in both pathways, and most of them belong to the COL family, which strengthens the findings of the role of the COL family in gastric adenocarcinoma.
	0.96	COL1A2, COL1A1, COL11A1, COL4A6, and THBS2) are involved in both pathways.
27171163	0.97	Col1A2, Col1A1, Col3A1, Col11A1 and Col15A1, which are commonly increased during fibrosis, beginning 2 weeks after IR exposure, was confirmed by real-time PCR analysis (Fig 1C and S1 Fig).
30948703	0.96	COL1A2, COL5A2, COL5A1, COL18A1, and COL11A1 belong to the collagen family, which comprises 28 members (I-XXVIII).
	0.89	COL1A2, COL5A2, COL11A1, COL18A1, FN1, and SPARC was associated with poor OS of GC patients.
	0.87	COL1A2, COL5A2, FN1, THBS1, COL5A1, SPARC, COL18A1, and COL11A1) were identified via PPI network analysis.
	0.59	COL1A2, COL5A2, COL11A1, COL18A1, FN1, and SPARC was related to poor OS in patients with GC.
28586059	0.96	COL1A2, COL3A1, COL5A2, COL6A1, COL6A2, COL6A3, COL11A1, COL12A1 and ITGA4 may interact with each other.
	0.95	COL1A2, COL6A2, COL6A1, ITGA4, COL1A1, COL11A1 and COL5A2) and neuroactive ligand-receptor interaction (dM3; P=9.49x10-4; Table IVB).
	0.92	COL1A2, COL3A1, COL5A2, COL6A1, COL6A2, COL6A3, COL11A1, COL12A1 and integrin alpha4 (ITGA4) may interact with each other in the dM2 module.
30542454	0.96	COL1A2, cathepsin K (CTSK), integrin subunit beta 4 (ITGB4), tissue inhibitor of metalloproteinases 1 (TIMP1), ITGA11, COL11A1, MYB proto-oncogene like 2 (MYBL2), karyopherin subunit alpha 2 (KPNA2), aurora kinase A (AURKA), TPX2, microtubule nucleation factor (TPX2) and cell division cycle 20 (CDC20) were identified as key upregulated genes in smoking-associated lung cancer (Fig. 2A and B).
	0.95	COL1A2, CTSK, ITGB4, TIMP1, ITGA11, COL11A1, MYBL2, KPNA2, AURKA, TPX2 and CDC20.
17411443	0.96	COL11A1, COL1A2 and COL5A2.
25860484	0.95	COL1A2, COL5A2, COL11A1, DSG3, ACHE, SERPINE1, SERPINB2, CXCL5, MMP1, PLAU, SPP1, GJB2, CLDN2, CDKN2A, CENPF, MAD2L1, and NCAM1), most of which (17 out of 18) were up-regulated in gastric cancer tissues (Fig 4).
	0.75	COL1A2, COL5A2, COL11A1, and SPP1) were most significantly altered and were all involved in the ECM-receptor interaction and focal adhesion pathway (Table 2).
	0.59	COL1A2, COL5A2, COL11A1, and SPP1) co-regulated by both TFs and miRNAs participated in ECM-receptor interaction and focal adhesion pathways.
28933415	0.95	COL11A1, COL1A2, COL3A1, COL4A2, and COL4A6) and cathepsins (CTSB); the latter is involved in an intricate activation cascade of proteases that leads to proteolysis, contrasting with the presence of overexpressed protease inhibitors (PI3, SERPINA3, SPINK1, SPINT1, ITIH4, and CSTB).
	0.94	COL11A1, COL1A2, COL3A1, COL4A2, COL4A6, FBLN5, FBN1, FGA, LTBP2, MATN4, SPINT1, VCAM1, and VTN) being increased in expression.
	0.81	COL11A1, COL1A2, COL28A1, COL3A1, COL4A2, COL4A6, DSP, EFEMP2, FBLN5, FBN1, HSP90AA1, HSPB1, LGALS1, LTBP2, MYL6, PI3, PKM, PRDX1, RNASE1, TGM4, UMOD, and VTN.
27356888	0.95	COL1A2, COL3A1, COL4A2, integrin alpha 1, COL4A1, biglycan and COL11A1.
31485619	0.94	COL11A1, COL1A1, COL1A2, COL5A1, COL5A2 and COL6A3, which was involved in the ECM-receptor interaction and PI3K-Akt signaling pathways.
29844680	0.93	COL11A1, COL4A1, COL5A2, COL5A1, COL1A1, COL1A2 and COL5A3) genes are collagen-coding genes, and the remaining one (THBS2) modulates collagen fibrillogenesis and plays a crucial role in the ECM assembly.
	0.90	COL11A1, COL4A1, COL5A2, COL5A1, COL1A1, COL1A2, COL5A3 and THBS2) and 46 edges with a score of 8.5 was detected by MCODE.
	0.63	COL11A1, COL4A1, COL5A2, COL5A1, COL1A1, COL1A2, COL5A3 and THBS2) was found.
30106150	0.93	COL1A2, COL5A2, COL10A1, COL11A1 and COL2A1, has the molecular function of extracellular matrix structural constituent.
	0.85	COL1A2, COL2A1, COL11A1 and SPARC, whose corresponding degree was beyond 10, were hub genes in the PPI network and closely related to cancer.
30428899	0.92	COL11A1, c MMP7, d ALB, e COL1A2, f COL3A1, g EGF, h FN1, i ITGA2, j SPARC, kTIMP1
	0.85	COL1A2, SPARC, COL3A1, TIMP1, COL5A1, COL11A1, and MMP7) with degree > 5 were screened as candidate genes.
25940674	0.91	COL1A2, COL2A1, COL3A1, COL4A1, COL5A1, COL11A1).
28938546	0.91	COL1A2, CLEC2B, ANGPT2, TCF4, MLLT10, COL11A1, FAR2 and SMEK1 (Table 1).
32050423	0.91	COL1A2, COL5A1, COL6A1, COL11A1, PLOD2, FGFR1, LDLRAD4, SMURF2, and SPARC was validated by qPCR analysis (Figure 4).
32194659	0.89	COL1A2, COL6A3, THBS2, COL5A2, COL11A1, FAP, MXRA5 and THY1 were upregulated in solid cancer tissues, and significantly associated with the overall survival of patients with cancer.
29285246	0.88	COL11A1, COL1A1, COL1A2), SULF1, CDH1, INHBA, VCAN as well as APOBEC3B played a vital role in the process of EC occurrence and development.
30002985	0.85	COL1A2, COL1A1, COL3A1, COL5A1, COL4A1, COL2A1, COL4A2, COL5A2, COL6A1, SERPINH1, COL6A3, COL11A1, COL12A1, COL10A1, COL8A1, FN1, SPARC, THBS1, FBN1, THBS2, ITGA5, ADAMTS2, TIMP1, BGN, and BMP1 (Fig. 5).
29724173	0.80	COL1A2, COL11A1, SLC26A2, COMP, COL9A1, and other genes.
30894224	0.64	COL1A2) and 1 upregulated DEGs (COL11A1) (Fig. 6).
20952505	0.62	COL11A1, OGN, COL14A1, COL1A2, DCN, ASPN, and CALD1) (Table 2).
27609069	0.62	COL1A2, FAP was also differentially expressed between inflamed/fibrotic tissues and cancer tissues although this difference in expression was not as prominent as for COL11A1 (compare Fig. S9B and Fig. 4B).
30884856	0.60	COL1A2, COL2A1, COL11A1).
19652764	0.57	COL11A1, COL1A1, COL3A1, COL1A2, COL15A1).
30828485	0.50	COL1A2, COL2A1, COL3A1, COL5A1, COL5A2, COL6A1, COL8A1, COL11A1, and COL24A1, may be candidate biomarkers of hydroxymethylation abnormalities in OA that may be considered for more accurate diagnosis and treatment of OA.