Publication for Col5a1 and Mmp2
| Species | Symbol | Function* | Entrez Gene ID* | Other ID | Gene coexpression |
CoexViewer |
|---|---|---|---|---|---|---|
| mmu | Col5a1 | collagen, type V, alpha 1 | 12831 | [link] | ||
| mmu | Mmp2 | matrix metallopeptidase 2 | 17390 |
| Pubmed ID | Priority | Text |
|---|---|---|
| 29615716 | 0.98 | Col5a1, Col1a1, Lamb2, Mmp14, Mmp2, Itgb3, Nid1, Igf1, and Cd44 formed a tight cluster within this network, stressing the importance of the establishment of the basement membrane and reorganization of endothelial cells as they acclimate following wound closure. |
| 0.98 | Col5a1, Col5a2, Col8a1, Col18a1, Nid1, and Lamc3, all of which encode for proteins involved in the maintenance of basement membrane, as well as Mmp14 and Mmp2, highlighting the persistent extracellular matrix remodeling. | |
| 0.86 | Col5a1, Col8a1, Mmp14, Mmp2, Sele, Cd44, Nid1, Eln, and Fbln5 encode for proteins that form interactions with strong confidence scores greater than 0.9 (Fig. 7d, Supplementary Data S5). | |
| 26023920 | 0.98 | COL5a1, ITGA4, VTN, MMP1a, MMP2 and MMP9 were upregulated in the bone healing process in comparison with the control samples (Fig 5). |
| 0.88 | COL5a1 and MMP2 levels presented a growing curve until 14d time period, followed by a decrease in its mRNA levels. | |
| 22802911 | 0.98 | COL5A1, COL5A2, COL5A3, COL7A1, COL8A1, MMP2, and ITGB1). |
| 23261783 | 0.98 | Col5a1, MMP-2, and MMP-9, indicating defective ECM transcription in the absence of MMP-28. |
| 24109192 | 0.98 | COL5A1, COL5A2, COL5A3, COL7A1, COL8A1, and MMP2, have been validated as direct targets of miR-29 by reporter gene assays. |
| 26589811 | 0.98 | MMP2 and the osteoclastogenic factor RANKL, in parallel with decreased expression of OPG and wound healing related factors (COL5A1, CTGF, CXCL11, FGF7, ITGA4, ITGA5, SERP1, VTN). |
| 27058578 | 0.98 | Mmp2), collagens (Col3a1, Col5a1, Col6a1, Col6a2, Col14a1 and Col15a1) and tissue inhibitor of metallopeptidase 1 (Timp1). |
| 29774125 | 0.98 | COL5A1, COL7A1) that are components of the extracellular matrix, as well as matrix metalloproteases that breakdown the extracellular matrix (MMP1, MMP2, MMP11, MMP14). |
| 31639302 | 0.98 | Col5a1, Col6a1, Ecm1, Emilin1, Fn1, Hapln1, Lamb3, Postn, Sparc, Spp1, Thbs1, Thbs2, Tnc), and 9 genes encoding modulators of remodeling (Adamts1, Adamts2, Ctgf, Mmp14, Mmp2, Tgfbi, Timp1, Timp2, Timp3) indicated that graphene foam provided a microenvironment conducive to expression of genes that are important in early chondrogenesis. |
| 26221607 | 0.97 | Col5a1, and Eln, which encode type I, III, and V collagen and elastin, but also downregulated MMP-2 and MMP-9 expression. |
| 31227742 | 0.97 | Col5a1, Col6a1) and extracellular matrix (ECM) remodeling (Timp1, Timp2, Mmp2, Pdgfa, Tgfb), see Fig. 5C. |
| 26891705 | 0.95 | COL5A1 is crucial for cardiovascular development; PLVAP helps to maintain vascular permeability of endothelial cells and MMP2 is a target of TGFbeta signalling and an important player in EMT. |
| 29348826 | 0.95 | Col5a1, Ecm1, Sparc, Col1a1, Col3a1, Lama3, and Lamb3) (Figure 7C, middle panel) and metalloproteinases (8 genes: Adamts2, Adamts5, Mmp3, Mmp2, Adamts8, Mmp11, Mmp14, and Mmp15) (Figure 7C, right panel), were also significantly down-regulated in the BLM+ApoJ group compared to those in the BLM+ViaJ group. |
| 19463168 | 0.83 | Mmp2), stathmin 1 (Stmn1), procollagens (Col5a1 and Col6a1), Wisp1 and the receptor tyrosine kinase Axl. |
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