Publication for Cntn1 and Negr1

Species Symbol Function* Entrez Gene ID* Other ID Gene
coexpression
CoexViewer
mmu Cntn1 contactin 1 12805 [link]
mmu Negr1 neuronal growth regulator 1 320840

Pubmed ID Priority Text
25061880 0.98 Cntn1, Ncam1, Negr1, Nrxn1 and Sh2b3 mRNAs and their respective long non-coding RNAs in an in vitro model of ischemic-reperfusion injury showed an inverse expression profile to the maturation process, thus suggesting their role(s) in maintaining neuronal structure and function.
0.98 Cntn1, Ncam1, Negr1 and Nrxn1 had differentially expressed mRNAs and lncRNAs associated with them and were also predicted to be targeted by the altered miRNAs (Figure 3A).
0.98 Cntn1, Ncam1, Negr1 and Nrxn1 which are responsible for neurite outgrowth and synaptogenesis (Figure 5, genes in bold).
0.98 Cntn1, Ncam1, Negr1, Nrxn1, Ntrk2 and Sh2b3 associated mRNAs and lncRNAs during ischemic injury could characterize the exact relationship between 7 mRNA-lncRNA targets (Table 3).
0.98 Cntn1, Ncam1, Negr1, Nrxn1, Sh2b3) was observed upon ischemic insult (Table 3).
0.97 Cntn1, Ncam1, Negr1, Nrxn1, Ntrk2 and Sh2b3) by both lncRNAs and miRNAs could be implicated in neuronal maturation.
0.97 Cntn1, Ncam1, Negr1 and Nrxn1 as well as Sh2b3 in the neurotrophin signalling pathway displayed a mainly inverse relationship between mRNA and antisense lncRNA gene pairs.
0.97 Cntn1, Ncam1, Negr1, Nrxn1, Ntrk2 and Sh2b3) that is altered during neuronal maturation (Figure 3), in an ischemic injury model (neuronal).
0.97 Cntn1, Ncam1, Negr1, Nrxn1) were targeted by all these miRNAs with miR-377 specifically targeting only the 4 cell adhesion molecules (Figure 5).
0.94 Negr1 and Nrxn1 was upregulated and expression of Cntn1 was downregulated upon maturation (Days 6 and 8).
0.87 Cntn1, Ncam1, Negr1, Nrxn1, Sh2b3 (Table 3).
29249937 0.95 NEGR1, OBCAM, and contactin-1 gradually increase during development and reach the highest level at 4 weeks after birth in the cerebral cortex, diencephalon, hippocampus, and cerebellum.
0.94 NEGR1, OBCAM, Ntm, Thy-1, LAMP, and contactin-1 as components of synaptic vesicles.



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