Publication for KRT81 and KRT86
Species | Symbol | Function* | Entrez Gene ID* | Other ID | Gene coexpression |
CoexViewer |
---|---|---|---|---|---|---|
hsa | KRT81 | keratin 81 | 3887 | [link] | ||
hsa | KRT86 | keratin 86 | 3892 |
Pubmed ID | Priority | Text |
---|---|---|
25287022 | 0.98 | K81, K85 and K86), we found differences in the rate of evolution, suggesting that these cetacean hair-type keratin genes may be under reduced selective constraints (P < 0.01, for all genes). |
0.96 | K81, K86, K83, K85, K84 and K82) and 21 other type II keratin genes (Figure 1). | |
0.96 | K81, K86 and K83 hair-type keratin genes were responsible for the hairless phenotype of the Hirosaki hairless rat. | |
0.95 | K81, K83 and K86 genes (Figure 3). | |
28785294 | 0.98 | K81, K83, K87 and K86 in the type II (Figs. 2 and 3). |
0.97 | K81-K86) expressed in hair follicle and hair which could directly affect hair growth. | |
26317522 | 0.98 | K81, K83, K85, and K86 (Fig 1). |
29371649 | 0.97 | K81, K82, K83, K84, K85, and K86; and type I K31, K33a, K33b, K34, K35, K32, K36, K37, and K38 (Supplementary Fig. S3a,b). |
0.94 | K81, K83, and K86) and group C (K82, K84, and K85). | |
0.84 | K81-K86). | |
0.54 | K81-87), and interestingly, deamidation was observed in keratin type I belonging to the families K33A, K33B, K34, and K35; type II belonging to families K82-K86; and several KAPs. | |
31847069 | 0.97 | K81-K86 (type II), K31-K40 (type I)), located both in the cortex and in the cuticle, representing the most important cells within the hair follicle involved in hair growth. |
0.93 | K81-K86 (former designation hHb1-hHb4), as reported in the experimental section. | |
0.86 | K81, K83, K85, and K86 (all types II hair keratin isoforms), increased cellular content was already detectable after 48 h of treatment. | |
0.82 | K81-K86) hair keratins expressed by hair follicular keratinocytes. | |
22393391 | 0.97 | keratin81 (NM_002281), keratin86 (NM_002284), Serpina3 (NM_001085), collagen type VIII alpha1 (NM_001850) (f.c.>5) and STAT1 (IFN-gamma signalling pathway) (NM_139266) (f.c.>2). |
26610607 | 0.97 | KRT81, KRT83 and KRT86) is upregulated in all resistant cells and its putative SE belongs to cluster 1 (Fig. 3b). |
21188018 | 0.96 | K81, K83, K86. |
24361867 | 0.96 | KRT86, KRT81 and KRT83 loci. |
28839397 | 0.96 | KRT81, KRT83, and KRT86 lead to autosomal dominant monilethrix whereas mutations in the desmoglein 4 gene cause an autosomal recessive form. |
30050820 | 0.96 | KRT81, KRT83, or KRT86, whereas in autosomal recessive form, mutation in the desmoglein-4 gene (DSG4) has been reported. |
31113963 | 0.96 | K81 - K86) with high-sulfur content. |
16831889 | 0.95 | K81-K86, led to last digit matching between the old (Table IV, column 1) and the new nomenclature (Table IV, column 4), so that this kind of "aide-memoire" exists for both types of human hair keratins. |
29784039 | 0.94 | K81, K83, and K86).. |
22628999 | 0.93 | KRT86 gene were identified in large families from Turkey and India as well as in the KRT81 gene in a family of Indian origin. |
32030317 | 0.92 | K81, K83 and K86 (spot 7)) compared to the Chinese and three keratins that were differently altered (K33b and two K86 (spots 7 and 8)) compared to the Malays. |
27284572 | 0.87 | KRT86, KRT83, and KRT81. |
23554671 | 0.82 | KRT81, KRT83 and KRT86. |
0.76 | KRT86 and KRT81 is thought to be a possible mutation hot spot in monilethrix. | |
0.73 | KRT86, KRT83 and KRT81 gene led to the detection of a heterozygous G to A point mutation in the KRT86 gene in the family (Fig. 4A). | |
0.57 | KRT86, as well as an HTM E413K mutation in hair keratin KRT81, have been reported. | |
0.55 | KRT81 and KRT83 and exon 1 of KRT86 among the patients. | |
32257198 | 0.82 | KRT81-KRT86) in each group of samples (Fig. 4). |
21176769 | 0.81 | K81 (Hb1) and K86 (Hb6) in the mid-cortex and shares an almost identical rod domain sequence, only one mutation in the 2B region (p.Glu407Lys) has been identified so far in a single monilethrix family with co-dominant manner. |
0.63 | KRT86 (previously KRTHB6), and HTM mutation in K86 (Hb6; 90%) as well as HTM mutation in K81 (Hb1;100%) have been identified. | |
30814526 | 0.76 | KRT81, and KRT86), intercellular junction associated proteins (DSG3, GJB2, JPH1, JPH2, PKP2, UPK2) and genes related to keratinocyte differentiation (IVL, EPPK). |
22716242 | 0.71 | K81, K83, K85 and K86, are expressed only in the hair shaft and follicles, and are responsible for keratodermas of the hair and nails (see www.interfil.org). |
24778539 | 0.65 | KRT86, KRT 81 and KRT 83 gene in autosomal dominant trait and mutation in desmoglein 4 was associated with autosomal recessive type of monilethrix. |
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