Publication for Tuba1a and Tubb2b

Species	Symbol	Function*	Entrez Gene ID*	Other ID	Gene coexpression	CoexViewer
mmu	Tuba1a	tubulin, alpha 1A	22142			[link]
mmu	Tubb2b	tubulin, beta 2B class IIB	73710

Pubmed ID	Priority	Text
21060993	0.97	TUBA1A, TUBB2B, and TUBB5 may play roles in DEX-induced inhibition of osteoblastogenesis.
	0.97	TUBA1A, TUBB2B, and TUBB5), IQGAP1, S100 proteins (S100A11, S100A6, S100A4, and S100A10), myosin proteins (MYH9 and MYH11), apoptosis and stress-related proteins (BAX, Hsp84b, Hsp60, PDCD6IP), and the downregulated protein levels of ATP synthase (ATP5O, ATP5H, ATP5A1, and ATP5F1), G3BP-1, and Ras-related proteins (Rab-1A, Rab-2A, and Rab-7) may be critical in the mechanism of the DEX-induced results.
	0.96	TUBA1A, TUBB2B, and TUBB5), IQGAP1, S100 proteins (S100A11, S100A6, S100A4, and S100A10), myosin proteins (MYH9 and MYH11), and apoptosis and stress proteins, while inhibited the protein levels of ATP synthases (ATP5O, ATP5H, ATP5A1, and ATP5F1), G3BP-1, and Ras-related proteins (Rab-1A, Rab-2A, and Rab-7) in MC3T3-E1 cells.
28388629	0.96	TUBA1A, TUBB2B, TUBB3 and TUBB5, whose roles include neuronal migration and axon guidance which impact on final brain structure and function, and where high expression levels are present in the adult or developing brain.
31086189	0.95	TUBA1A, TUBB2B, TUBB3) and microtubule-associated proteins (LIS1, DCX, KIF2A, KIF5C, DYNC1H1) have been linked to a large spectrum of cortical malformations.
29962938	0.94	TUBA1A and TUBA8, as well as the beta-tubulin genes, TUBB2B and TUBB3, have all been identified in connection with PMG (Abdollahi et al.,; Jaglin et al.,; Poirier et al.,).
25911067	0.93	TUBA1A, TUBB2B).
27594048	0.89	TUBA1A, TUBA8, TUBB/TUBB5, TUBB2A, TUBB2B, TUBB3, and TUBG1 have all been associated with CNS defects such as polymicrogyria, lissencephaly, cortical dysplasia, and congenital fibrosis of the extraocular muscles .
31386652	0.87	Tubb2b, however, loss of Tuba1a is catastrophic for the mouse embryo.
	0.86	(TUBA1A) ; TUBULIN, BETA-2A (TUBB2A); TUBB2B; TUBB3; TUBB4A; and TUBB/TUBB5.
	0.68	Tubb2b, Tuba1a is absolutely required for survival to weaning as we did not recover any homozygotes for either Tuba1a deletion allele (n = 177 total; Table 2).
	0.63	TUBA1A and TUBB2B account for ~5% and ~1.2% of lissencephaly and related malformations of cortical development, respectively.
26299390	0.75	Brdp (+/-) show hyperactivity like Tuba1a heterozygotes.