Publication for Xpo1 and Cse1l
Species | Symbol | Function* | Entrez Gene ID* | Other ID | Gene coexpression |
CoexViewer |
---|---|---|---|---|---|---|
mmu | Xpo1 | exportin 1 | 103573 | [link] | ||
mmu | Cse1l | chromosome segregation 1-like (S. cerevisiae) | 110750 |
Pubmed ID | Priority | Text |
---|---|---|
25948791 | 0.98 | XPO2-7), XPO1/CRM1 is the sole nuclear exporter for some of the major tumor suppressors (i.e., p53), cell cycle regulators (i.e., CDKN1a) and growth promoting proteins (i.e., survivin). |
24490135 | 0.97 | XPO1, XPO4, and Cse1L were also more highly expressed in mESCs compared to MEFs. |
27232334 | 0.97 | Xpo1 and Xpo5 are additional members of the exportin family, have some structural similarities to Xpo2 and mediate the export of proteins with a nuclear export signal and of RNA respectively. |
20886002 | 0.95 | Exp1 and Exp2, therefore reflecting the impact of CLA on adipose miRNAs. |
0.94 | Exp1 and Exp2). | |
0.86 | Exp1 and only the highest dose of CLA in Exp2 (CLA4) produced a significant increase of miR-221 with respect to both CLA3 and control groups (P<0.01). | |
0.81 | Exp1 and, despite moderate hyperglucemia and hyperinsulinemia in Exp2, insulin sensitivity, assayed by the revised quantitative insulin sensitivity check index is preserved in this experimental setting. | |
28332554 | 0.95 | EXP 1 (i.e., discontinuous oral feeding with fucoidan) exhibited reduced efficacy in inhibiting tumor growth compared with that of the EXP 2 continuous fucoidan treatment. |
0.71 | EXP 2 were significantly reduced compared with those of the mice in EXP 1 (Fig. 1B and C). | |
29379507 | 0.95 | Exp#1, and p = 0.027 in Exp#2; Figure 4D). |
24068922 | 0.93 | Exp 1: F2,16 = 6.0, p = 0.011, Exp 2: F2,25 = 21, p<0.001) as well as the density of resistant transmission stages (Fig. 2IJ). |
0.86 | Exp 1: F2,16 = 7.3, p = 0.006, Exp 2: F2,25 = 34, p<0.001). | |
0.73 | Exp 1: F2,16 = 15, p<0.001, Exp 2: F2,25 = 29, p<0.001). | |
30515275 | 0.92 | Exp-1 and Exp-2 could be explained by the following; (1) timing to start AfGAE administration, (2) tissue-specificity of certain cytokine productions, and (3) different sensitivity of certain cytokine production by local inflammation vs. systemic inflammation due to aging. |
0.85 | Exp-1 (P < 0.001), whereas this dose-dependency phenomenon was not observed in a treatment study, Exp-2. | |
26637797 | 0.87 | Exp 1) was decreased by 35% by rAAV-GFP-miRC9 (n=4, one-way ANOVA, Bonferroni's multiple comparison, p<0.05), and 24% in the pooled embryo cultures in Exp 2 (not statistically significant; n=3, one-way ANOVA, Bonferroni's multiple comparison). |
31990271 | 0.87 | Exp1 and Exp2 for proficient mice and this decrease is larger for proficient compared to naive mice (compare naive vs. proficient in Figure 7Bi and ii). |
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